DMT

Common Names: N-Dimethyltryptamine, N-DMT, Dmitri, The, Spirit

Psychoactive Effects

Positive Effects

  • Euphoria

Neutral Effects

  • Intense visual hallucinations
  • Complete reality replacement
  • Entity encounters
  • Time dilation/compression
  • Ego dissolution
  • Transcendental experiences

Negative Effects

  • Anxiety
  • Fear

Dosage Guidelines

Route Threshold Light Common Strong Heavy
Smoked 2-5 mg 5-15 mg 15-30 mg 30-50 mg 50+ mg
Oral 20-50 mg 50-100 mg 100-200 mg 200-300 mg 300+ mg

Note: DMT is naturally occurring in many plants and animals. When taken orally, it requires a monoamine oxidase inhibitor (MAOI) to be active. The experience is often described as the most intense psychedelic experience possible.

Duration

Phase Duration
Onset Immediate (smoked)
Peak 2-5 minutes
Total 5-15 minutes
After effects 15-30 minutes

Pharmacology

Mechanism of Action: DMT acts as a partial agonist at serotonin 5-HT2A receptors, similar to other classical psychedelics. It also has affinity for 5-HT1A, 5-HT2B, 5-HT2C, and sigma-1 receptors.

Metabolism: Rapidly metabolized by monoamine oxidase (MAO) enzymes, particularly MAO-A. When taken orally, it requires MAO inhibitors to prevent rapid breakdown.

Half-life: Very short, approximately 10-15 minutes when smoked/vaporized.

Bioavailability: ~50% when smoked/vaporized, but essentially 0% when taken orally without MAO inhibitors due to rapid metabolism.

Risks and Harm Reduction

DependenceToxicityWithdrawalAddictionOverdose

⚠️ Important: Always practice harm reduction. Start with low doses, test your substances, and ensure a safe set and setting. Never use alone.

Legal Status

Disclaimer: Legal status varies by jurisdiction and may change. This information is for educational purposes only.

References

  • • Strassman, R. J. (2001). DMT: The Spirit Molecule. Park Street Press.
  • • Nichols, D. E. (2016). Psychedelics. Pharmacological Reviews, 68(2), 264-355.
  • • Carbonaro, T. M., & Gatch, M. B. (2016). Neuropharmacology of N,N-dimethyltryptamine. Brain Research Bulletin, 126, 74-88.
  • • McKenna, D. J., et al. (1984). Monoamine oxidase inhibitors in South American hallucinogenic plants: tryptamine and beta-carboline constituents of ayahuasca. Journal of Ethnopharmacology, 10(2), 195-223.
  • • Information compiled from PsychonautWiki